The development rule of type 2 diabetes

Type 2 diabetes, including islet dysfunction and insulin resistance in two ways.

Islet dysfunction in type 2 diabetes is characterized by islet β-cell damage is minor damage from the gradual increase, the ability to secrete insulin gradually decreased.

Insulin resistance is the body's liver, muscle, fat tissue cells such as the role of insulin resistance, so that insulin can not properly play its role. Manifested in two aspects: the ability of insulin to transmit blood glucose and insulin to reduce the inhibition of hepatic glucose output decreased.

The role of insulin receptor-deficient so that the effectiveness of fall - can not be successfully opened the glucose-insulin-channel, glucose can not be successfully enter into the cells, which is the so-called insulin resistance.

The second aspect of insulin resistance is manifested in the liver: insulin on hepatic glucose output has in vitro. Under normal circumstances, when blood sugar rises, insulin secretion increased, so the inhibition of liver glucose output. But when there is "insulin resistance", the insulin regulation of hepatic glucose output "failure", it can not effectively suppress hepatic glucose output.

Insulin resistance is characterized by abnormal elevation of the need for insulin. Increase the extent of your requirements and the amount of insulin, insulin resistance and islet function.

About 70% ~ 90% in patients with type 2 diabetes is insulin resistance and insulin secretion by the lack of double factors. Insulin resistance is the "cause" injury of islet function is "fruit." No islet dysfunction, how much insulin, islet function will give you the number of insulin secretion, they will not get diabetes. Insulin secretion can not be long-term, unlimited increase in insulin secretion will eventually reach the limit and can not continue to increase. If the long-standing insulin resistance, pancreatic functional reserve will vanish; and because of pancreatic islet β-cells secrete insulin will not be able to meet the needs of the body to insulin increases continue to increase indefinitely, that is, can not meet the needs of the body. Emergence of the "relative lack of insulin" - the body's insulin levels higher than normal, but excessive insulin need, has been inadequate, and blood sugar will rise. With the extension of course, the damage islet function will become increasingly heavy secretion of insulin would be less and less, it appears the lack of insulin - your insulin levels even lower than normal.

The law of development of type 2 diabetes:

(1) insulin resistance phase: the so-called insulin resistance is the organization of the body less sensitive to insulin. The body to maintain normal blood sugar, the need for more insulin than normal, the body will be through the use of the reserve part of the islet to produce more insulin, which is the so-called hyperinsulinemia.

(2) islet dysfunction stages: a gradual increase of this damage process. Effect of early pancreatic islet dysfunction light, the lack of insulin effect of light, the secretion of insulin in some states can meet the body's needs. As the disease worsened islet dysfunction gradually worsened, the secretion of insulin the body will not be able to meet the needs of a variety of state, performance has continued to rise for the fasting blood glucose.

(3) islet failure stages: pancreatic β-cells, further serious damage, failure will be developed to the stage. The body would be a serious lack of insulin, if not timely replenishment of exogenous insulin, it will endanger human life.